Zika virus RNA is frequently present in the semen of men with symptomatic ZIKV infection and can persist for over 6 months.
Pregnant women may have about a 3-fold longer estimated median detection window for Zika virus RNA in serum.
A vaccine against Zika virus, once available and effectively deployed, may substantially reduce prenatal infections.
A Zika virus vaccine shown to be of moderate to high efficacy in eliminating prenatal infections provides 90% coverage.
Researchers estimated this risk among pregnant women with symptomatic Zika virus infection in French territories in the Americas.
Prompt identification may help reduce the risk for subsequent renal failure and minimize overall disease burden.
Animal model revealed a potential relationship between Zika virus infection and uteroplacental pathology that appears to affect oxygen delivery to the fetus during development.
A total of 5168 noncongenital Zika virus disease cases were reported from US states and the District of Columbia in 2016.
There was an increase in the number of birth defects potentially related to Zika virus infection from the first half of 2016 to the second half of 2016.
Although Zika virus is known to be transmitted by mosquitos, sexual contact, blood transfusion, or laboratory exposure, there is also evidence supporting maternal transmission to infants via breastfeeding, delivery, or close contact.
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